肌I弓蛋白（Troponins)是診斷急性心肌梗塞的重要標記。I型肌#5蛋白（Tnl)是存在於心臟肌肉細胞内之調節蛋白質，因此當心肌細胞受損時，血清中肌鈣蛋白數値便會昇高。實證醫學顯示，雖病人心電圖未呈現STT節段上昇，若檢測血清I型肌鈣蛋白昇高時，在診斷急性心肌梗塞之正確率達100%，而檢測血清T型肌鹤蛋白昇高，則診斷正確率達 75%。醫師平時針對一氧化碳中毒患者之醫療處置，若患者沒有胸部不適，或心電圖STT節段的無異常變化，並不會常規檢測血清I型弓蛋白。本文爲探討一名20歲原身體健康狀況良好的軍人，因懷疑一氧化碳中毒，撿測血清中I型肌麫蛋白上昇，而診斷爲急性心肌梗塞。特提出報告，藉能拋磚引玉，有更大規模研究來確立嚴重一氧化碳中毒病患，檢驗肌1弓蛋白的重要性。

Myocardial injury can be identified by elevated serum level of troponin, a regulatory protein of thin actin filaments of the cardiac muscle. In patients with inconclusive electrocardiogram (ECG) finding (i.e., no ST-segment elevation), a positive troponin I (Tn I) is 100% and troponin T (Tn T) is 75% predictive of acute myocardial infarction. In daily practice, physicians would not routinely check serum Tn I level in patients suffering from carbon monoxide (CO) intoxication if patients do not complain of chest discomfort or when ECG examination shows no significant ST-segment changes. We report a 20-year-old previously healthy soldier with CO intoxication. Acute myocardial infarction was diagnosed by elevated serum Tn I that was further supported by typical serial cardiac enzyme changes, and severe systolic hypokinesia of left ventricle in echocardiogram. This case suggests the importance of measuring serum Tn I level to early disclose myocardium injury in patients with CO intoxication.